Mice lacking inward-rectifying potassium channel Kir6.1
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United States of America Patent
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Oct 3, 2006
Grant Date -
May 20, 2004
app pub date -
Mar 14, 2003
filing date -
Mar 19, 2002
priority date (Note) -
Expired
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Abstract
Mice homozygous for the lack of inward-rectifying potassium channel Kir6.1 gene are disclosed. The mice causes a high incidence of sudden death associated with arrhythmia (atrioventricular block) caused by spontaneous cardiac ischemia, a condition similar to Prinzmetal angina (variant angina) in human, representing an animal model of Prinzmetal angina. Mice heterozygous for the lack of inward-rectifying potassium channel Kir6.1 gene are also disclosed, which are used as parent mice for reproduction of the homozygous mice.
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